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Dr Anne Colville

Image of Anne Colville
Research Associate, School of Civil and Environmental Engineering
Associate Member, CENS - Centre for Environmental Sustainability
B.Sc (Hons) (Sydney U), M. Sc. (UTS), M.Sc. (Uni. Melb), PhD (UTS)
 
Phone
+61 2 9514 7841

Journal articles

Mohammed Abdul, J., Colville, A.E., Lim, R.P., Vigneswaran, S. & Kandasamy, J.K. 2012, 'Use of duckweed (Lemna disperma) to assess the phytotoxicity of the products of Fenton oxidation of metsulfuron methyl', Ecotoxicology And Environmental Safety, vol. 83, pp. 89-95.
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Because of pressure on water supplies world-wide, there is increasing interest in methods of remediating contaminated ground waters. However, with some remediation processes, the breakdown products are more toxic than the original contaminant. Organic matter and salinity may also influence degradation efficiency. This study tested the efficiency of Fenton oxidation in degrading the sulfonylurea herbicide metsulfuron methyl (MeS), and tested the reaction products for phytotoxicity with the Lemna (duckweed) bioassay. The efficiency of degradation by Fentonâs reagent (Fe2þ ¼0.09 mM; H2O2¼1.76mM, 4 h) decreased with increasing initial MeS concentration, from 98% with 5 mg/L MeS, to 63% with 70 mg/L MeS. Addition of NaCl (10 mM) and organic matter (humic acid at 0.2 and 2.0 mg C/L as Total Organic Carbon) reduced the efficiency of degradation at low initial MeS concentrations (5 and 10mg/L), but had no effect at high concentrations. The residual Fentonâs reagent after Fentonâs oxidation was toxic to Lemna. After removal of residual iron and H2O2, the measured toxicity to Lemna in the treated samples could be explained by the concentrations of MeS as measured by HPLC/UV detection, so there was no evidence of additional toxicity or amelioration due to the by-products or formulation materials.
Pablo, F., Krassoi, R., Jones, P.R., Colville, A.E., Hose, G.C. & Lim, R.P. 2008, 'Comparison of the fate and toxicity of chlorpyrifos - Laboratory versus a coastal mesocosm system', Ecotoxicology And Environmental Safety, vol. 71, no. 1, pp. 219-229.
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The widespread use of chlorpyrifos for pest control in urban and rural environments poses a risk of contamination to aquatic environments via runoff, spray drift or spillage. The aim of this study was to assess the fate of chlorpyrifos and its toxicity t
Colville, A.E., Jones, P.M., Pablo, F., Krassoi, R., Hose, G.C. & Lim, R.P. 2008, 'Effects of chlorpyrifos on macroinvertebrate communities in coastal stream mesocosms', Ecotoxicology, vol. 17, no. 3, pp. 173-180.
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This study measured the effects of a single pulse of chlorpyrifos at nominal concentrations of 1 and 10 mu g/l on the macroinvertebrate community structure of a coastal stream mesocosm system. Analysis of data using Principal Response Curves (PRC) and Monte Carlo tests showed significant changes in the treated stream mesocosms relative to that of the controls. These changes in the macroinvertebrate assemblages occurred within 6 h, and persisted for at least 124 days after dosing. Significant community-level effects were detected at the lowest concentration on days 2 and 16 post-dosing, giving a no-observed effect concentration (NOECcommunity) of 1.2 mu g/l (measured). The mayflies Atalophlebia sp. and Koorrnonga sp., Chironomidae and Acarina were all sensitive to chlorpyrifos and decreased in abundance in treated mesocosms after dosing. The fauna of these coastal stream mesocosms showed similar sensitivity to chlorpyrifos with that of other reported studies, but there was no evidence of recovery after 124 days.
Colville, A.E. & Lim, R.P. 2003, 'Microscopic structure of the mantle and palps in the freshwater mussels Velesunio ambigus and Hyridella depressa (Bivalvia : Hyriidae)', Mulluscan Research, vol. 23, pp. 1-20.
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Burnstock, G., Campbell, G., Satchell, D. & Smythe, A. 1997, 'Evidence that adenosine triphosphate or a related nucleotide is the transmitter substance released by non-adrenergic inhibitory nerves in the gut. 1970.', British journal of pharmacology, vol. 120, no. 4 Suppl, pp. 337-357.
Sneddon, J.D., Smythe, A., Satchell, D. & Burnstock, G. 1973, 'An investigation of the identity of the transmitter substance released by non-adrenergic, non-cholinergic excitatory nerves supplying the small intestine of some lower vertebrates', Comparative and General Pharmacology, vol. 4, no. 13, pp. 53-60.
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1. 1. Non-adrenergic, non-cholinergic excitatory postganglionic fibres in the splanchnic nerves supply the small intestine of the toad (Bufo marinus) and the lizard (Tiliqua rugosa). 2. 2. 5-Hydroxytryptamine, histamine, bradykinin, and prostaglandin E1 have been rejected as the putative neurotransmitters in these nerves on the grounds that they do not have actions which parallel those of nerve stimulation or that drugs which antagonize their effects do not similarly antagonize the effects of nerve stimulation. 3. 3. ATP produces excitatory responses, which closely mimic those of non-adrenergic, non-cholinergic nerve stimulation. Quinidine causes parallel block of the responses to nerve stimulation and exogenously applied ATP, but this action may be non-specific. 4. 4. While the results do not provide direct evidence for purinergic excitatory nerves to the small intestine of lower vertebrates, they are consistent with the hypothesis. © 1973.
BURNSTOCK, G., SATCHELL, D.G. & SMYTHE, A.N.N.E. 1972, 'A comparison of the excitatory and inhibitory effects of non-adrenergic, non-cholinergic nerve stimulation and exogenously applied ATP on a variety of smooth muscle preparations from different vertebrate species', British Journal of Pharmacology, vol. 46, no. 2, pp. 234-242.
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BURNSTOCK, G., DUMSDAY, B. & SMYTHE, A. 1972, 'Atropine resistant excitation of the urinary bladder: the possibility of transmission via nerves releasing a purine nucleotide', British Journal of Pharmacology, vol. 44, no. 3, pp. 451-461.
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BURNSTOCK, G., CAMPBELL, G., SATCHELL, D. & SMYTHE, A.N.N.E. 1970, 'Evidence that adenosine triphosphate or a related nucleotide is the transmitter substance released by non-adrenergic inhibitory nerves in the gut', British Journal of Pharmacology, vol. 40, no. 4, pp. 668-688.
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